First reference (dated an ancient 1985):
to the tendency for subsequent development of neurofibrillary tangles or outright Alzheimer's disease in individuals with industrial exposure to aluminum have not, to our knowledge, been published
Despite its natural abundance, it is clear that aluminum can be
a potent neurotoxin to certain mammalian species. The means
by which the element gains access to the tangle-bearing neurons seen in the brains of Alzheimer's disease victims or of the affected Chamorro natives currently remains unknown
It's only speculative, exactly zero evidence yet! And the speculation isn't that plausible, too.
Second reference (dated 2000):
"The monthly intracisternal
inoculation of aluminum chloride (AlCl3) ... receiving either single (1000 microg) or repeated sublethal (100 microg monthly) ... Our studies demonstrate that microglial activation in vivo is inhibited by AlCl3 exposure, and that a correlation exists between the extent of microglia suppression and the potential for recovery. This suggests that microglial activation is an important determinant of neuronal injury."
1) The aluminium was injected directly into the smallish brains of those poor rabbits and check the numbers - these are more than billion times higher than any thinkable exposure by deodorant and then the rabbits eventually recovered.
2) The focus of the study was totally differnt from checking for Alzheimer Desease.
3) The origin of Alzheimer Desease is also related to other factors, and maybe it is not too different from scrapie or later BSE, induced by prions, which became quite popular in the early 80s (paralled by AIDS).
I'm quite engaged with this because my physician suggested I should take a Lipobay medicamentation because of my sky rocketing cholesterine levels. If I did I would be plain dead now. From that my sceintific education was worth the effort. In made me use my brain (not demented yet) and being critical against authorities.